节律性闪光刺激提高EEG阳性率的研究附109例临床EEG闪光刺激分析

本文通过１０９例节律性闪光刺激临床脑电图的研究，探讨：１．正确使用间歇性节律性闪光刺激提高ＥＥＧ的阳性率；２．研究了闪光后的节律同化问题；３．发现症状性头痛“Ｈ”反应占９０％；４．毫针蝶骨电极加闪光刺激可以提高阳性率７５％。     

苯酮尿症的临床及脑电图研究

本文报告18例苯酮尿症,男女各9例,均具典型临床表现,尿三氯化铁试验均呈阳性反应,9例行苯丙氨酸水平测定,均明显高于正常水平。全部病例接受了EEG检查,其结果显示了脑电图异常的式样似与年龄及癲痫发作类型有关。文内对接受低苯丙氨酸饮食治疗且连续在本院随诊的12例病人的预后进行分析,强调尽早开始饮食治疗的重要性。     

Clonazepam对癫痫儿童脑电图的作用

作者对80例癫痫患者(包括两名青年人)于静脉注射 Clonazepam(每公斤0.03～0.3毫克)前、中、后描记脑电图。各例都有β活动,26例变为正常,另26例脑电图显著改善,1例脑电图更坏,27例脑电图无改变。原脑电图4例正常,24例有局灶性尖波棘波,6例有局灶性棘波及慢波,37例     

苯丙酮尿症患者头发微量元素锌、铅初步分析

苯丙酮尿症是先天性代谢缺陷病,为常染色体隐性遗传,至今尚无特效治疗。且于苯丙氨酸羟化酶的缺陷,血液中苯丙氨酸含量升高,尿中排出大量苯丙酮酸、苯丙氨酸和苯乙胺。患者表现不同程度的智力衰退,生长延缓和癫痫发作,皮肤皮皙,头发金黄,尿有鼠臭。据报道头发能反映机体微量元素的状态,不同颜色头发中微量元素的含量不同,我们应用质子激发X线分析法,对17名患儿和15名性别健康儿童头发样品进     

晚发性癲痫

本文对255例晚发性癫痫进行回顾性研究。各种不同病因所占比例分别为:头部外伤28例(10.9％),CNS感染18例(7.1％),脑血管病16例(6.3％),颅内占位病变14例(5.5％),脑囊虫病10例(3.9％),不同原因的颅内钙化9例(3.5％),代谢障碍6例(2.4％),其它原因(先天畸形、缺氧性脑病等)17例(6.7％),原因不明137例(53.7％)。文中对临床病史、症状及体征、EEG异常及颅脑CT扫描进行比较分析,认为在前三者的基础上再考虑是否进行CT扫描检查。     

癫痫发作间期脑电图SPECT脑显象脑CT的比较(论著摘要)

对人脑的研究继电生理及多种形式检查后又有新的进展,如正电子发射计算机断层(PET)及单光子发射计算机断层(SPECT)。~(99m)Te-HM-PAO能穿过血脑屏障,在脑组织的聚集量与血流量成正比。断层图象示大脑皮层灰质,基底节,丘脑及小脑为高放射性浓聚后。癫痫病灶发作时血流灌注增加,发作间期血流灌注减少。利用此特点国外为顽固部分复杂癫痫者作病灶定位。从而进行较满意的手术治疗。本研究记录分析了40例癫痫患者进行~(99m)-Te-HM-PAO脑断层显像检查的结果。其中四例全身型强直阵挛发作三者均未发现异常。二例儿童良性癫痫的脑电图示中央,中颞尖波灶,SPECT脑显象正常。34例P分复杂发     

Cobb氏顶部局灶性theta节律

Cobb氏theta节律乃持续或阵发的40～70μv,每秒5～9周波正弦或尖波节律,见于大脑后部导联,是一罕见现象,Cobb认为它有诊断意义。临床及病理均证实患者有中央和顶回大脑半球表面或纯在皮质下病变,多为脑膜瘤。本文报道我们30余年所仅见3例具有Cobb氏thcta节律的患者,但都不是脑膜瘤。在我们的脑瘤,特别是欠状窦旁脑膜瘤病例中,并未见到Cobb氏theta节律,这可能是因它容易为同时存在的delta波所掩盖以致漏诊。我们认为病因不只限於脑膜瘤,典型的Cobb节律是病理改变,有其诊断意义。     

头针对脑电图影响的初步观察

<正> 据报道,头针疗法对于神经系统疾病的疗效很突出,并在脑电图的研究中发现头针“对皮层功能的调节作用和病理状态的恢复作用”。作者还口头报道有“头针波”的发现。在医疗实践中,我们也观察到头针治疗的某些疗效。本工作首先试图验证上述脑电图研究报道的可靠性,以便进一步深入研究。     

WILSON'S DISEASE (HEPATOLENTICULAR DEGENERATION CLINICAL ANALYSIS OF 80 CASES

80 typical cases of Wilson's disease were observed over 54 years, 58 0f whom were hos- pitalized, an incidence of 19.7 per 100,0'00 general hospital inpatients and 522 per 100,0001 neurologic inpatients. The average age of onset was 23.5 years. Average age of onset before 15 years was 50To, between 20-30 26.91To and before 30 76.5.9To. 51.25'Io of our 80 patients had first involvement of the nervous system commencing at an average age of 25.2 years and 95To exhihited neurologic symptoms and signs. 39.99To had primary onset of hepatic disorder at an average age of 14.2 years and 80% showed evidence of hepatic dysfunction. 3.99To had renal involvement as first manifestation of disease and 40V/o showed evidence of renal damage. Aminoaciduria was demonstrated in 6.25%, 0.5'/o had affection of bones and joints, 2.5'/o had mental sympt.oms as first manifestation of the disease and 46.25% showed mental symptoms. 3 showed evidence of heart involvement. EEG findings and study of copper content of brain and liver wiU be pre- sented in other papers. Autopsy was performed on 5 with typical findings of Wilson's disease. Copper oxidase activity was lowered in 95%. Kayser-Fleischer ring was found in 73.75%. Urinary copper content was high in 96.97To (32/33 cases). Serum copper content was low in 65.22T。 (15;23 cases). 62 0f our 80 patients (77.50To) had well documented family histories. It was posi- tive in 10 patients of 9 families and highly suspected in another 4 in agreement with auto- somal recessive heredity. With regard to pro- gnosis 11 patients remain apparently healthy, 38 continue working or studying while taking treat- ment, 24 are invalids and 7 died. It appears that the longer the interval between onset, of disease and diagnosis, the worse the prognosis, indicat- ing the importance of early diagnosis and treat- ment.