A reexamination of plasticity-induced crack closure in fatigue crack propagation

The crack closure concept is often used to consider the R-ratio and overload effects on fatigue crack growth. The presumption is that when the crack is closed, the external load produces negligible fatigue damage in the cracked component. The current investigation provides a reassessment of the frequently used concept with an emphasis on the plasticity-induced crack closure. A center cracked specimen made of 1070 steel was investigated. The specimen was subjected to plane-stress mode I loading. An elastic–plastic stress analysis was conducted for the cracked specimens using the finite element method. By applying the commonly used one-node-per-cycle debonding scheme for the crack closure simulations, it was shown that the predicted crack opening load did not stabilize when the extended crack was less than four times of the plastic zone size. The predicted opening load was strongly influenced by the plasticity model used. When the elastic–perfectly plastic (EPP) stress–strain relationship was used together with the kinematic hardening plasticity theory, the predicted crack opening load was found to be critically dependent on the element size of the finite element mesh model. For R = 0, the predicted crack opening load was greatly reduced when the finite element size became very fine. The kinematic hardening rule with the bilinear (BL) stress–strain relationship predicted crack closure with less dependence on the element size. When a recently developed cyclic plasticity model was used, the element size effect on the predicted crack opening level was insignificant. While crack closure may occur, it was demonstrated that cyclic plasticity persisted in the material near the crack tip. The cyclic plasticity was reduced but not negligible when the crack was closed. The traditional approaches may have overestimated the effect of crack closure in fatigue crack growth predictions.