慢性硬膜下血肿31例诊疗体会

目的探讨单纯钻单孔引流术治疗慢性硬膜下血肿的可行性、实用性、有效性。方法31例慢性硬膜下血肿患者均行单纯引流钻孔术治疗。结果31例患者均临床治愈出院,随访6月～1年,仅3例遗留少量硬膜下积液,未行特殊治疗,1年后自愈。其余病人均无明显后遗症,术后患者生存质量均恢复到发病前水平。结论这种手术方法简单,并发症少,疗效满意,是治疗慢性硬膜下血肿的一种有价值的方法。     

机化型慢性硬膜下血肿1例

正慢性硬膜下血肿(chronic subdural hematoma,CSDH)好发于中老年人。机化型CSDH(organized chronic subdural hematoma,OCSDH)血肿腔内混杂有条索状的分隔,血肿周围有增厚的包膜包裹,部分病例血肿腔内膜有钙化形成,有报道称之为"铠甲脑"。本文报道2018年3月收治的1例OCSDH。1病例资料91岁男性,因间断性头痛、头晕半年余,加重伴左侧肢体活动障碍1周就诊。头部外伤史年余,当时无症状,未在意,无癫痫发作。入院时体格检查:神志清楚,精神稍差,言语对答尚流利,左侧肢体肌力Ⅲ级,病理反射阴性。头颅CT表现     

慢性硬膜下血肿96例诊治分析

我科自1998—01～2009—10共收治CDSH病人96例,行血肿钻孔引流术,效果良好,现总结如下。     

慢性硬膜下血肿患者的临床预后和复发率分析

目的分析慢性硬膜下血肿（CSDH）患者的临床预后及复发率。 方法回顾性分析青岛市西海岸新区人民医院神经外科自2012年6月至2017年12月收治的117例CSDH患者的一般资料、CT表现、血肿量及手术方式,分析其预后复发的情况。 结果本次入组117例患者,平均随访3个月,104例患者预后良好,13例患者复发。单因素分析显示患者年龄、术前CT分型和血肿量差异有统计学意义（P<0.05）,手术方式对预后复发的影响差异无统计学意义（P>0.05）;多因素Logistics回归分析进一步发现患者年龄、术前CT影像及血肿量与患者血肿的复发相关。 结论CT分型可作为CSDH手术方式和判断预后的重要指标,根据术前CT征象以及患者的病情选择最优的治疗方案,降低血肿复发率。     

双腔引流管治疗小儿慢性硬膜下血肿（附28例报告）

我院自1997年3月至2001年11月共对28例小儿慢性硬膜下血肿(CSDH)采用钻孔双腔引流管引流,效果显著。1临床资料1.1一般资料小儿CSDH28例,其中男18例,女10例,最大年龄9岁,最小1岁5个月,平均6.2岁。所有28例患儿均经CT检查,中线结构移位20例,脑室受压变形18例。1.2治疗方法和结果所有28例均采用钻孔引流术。术后留置双腔引流管(江苏省扬州市邗江双菱医疗器械厂生产,内套管为冲洗管,外套管为带侧孔的引流管)引流。一管接抗菌盐水持续滴入,另一管接无菌引流袋。抗菌盐水为生理盐水500ml加庆大霉素8万IU或先锋V号1.0g,滴入速度为6~8滴/min,…     

慢性硬膜下血肿的临床分析——附45例报告

慢性硬膜下血肿是颅内血肿的一种类型,临床上较为多见。我院1989年4月～1995年4月共收治患者45例,报告如下。 一、临床资料 (一)一般情况 45例中男性41例,女性4例;年龄小于20岁者3例,20～50岁者11例,大于50岁者31例:有明显外伤史者39例,发病时间25天～2年半,     

钻孔引流术在慢性硬膜下血肿的应用

慢性硬膜下血肿（chronic subdural hematoma,CSDH）系属外伤3w以后出现症状,位于硬脑膜与蛛网膜之间,具有包膜的血肿。CSDH是神经外科常见病症之一,好发于老年人和儿童,占颅内血肿的10％,占硬脑膜下血肿的25％,其中双侧血肿的发生率高达14％。慢性硬膜下血肿的治疗以颅骨钻孔引流术为首选,创伤小。我科2006年2月至2008年7月共收治62例慢性硬膜下血肿患者,采用钻孔治疗的方法,取得很好的效果。总结如下。     

钻孔引流术治疗慢性硬膜下血肿临床体会

<正>慢性硬膜下血肿(chronic subdural hematoma,CSDH)是神经外科常见病症之一,好发于中老年人,首选治疗方法是钻孔引流术[1];我院自2011年1月至2013年7月收治CSDH患者93例,经钻孔引流治疗效果满意,现报告如下。一、对象与方法1.一般资料:本组93例,其中男63例,女30例,年龄32~76岁,平均56.8岁,有明确外伤史73例,否认外伤史17     

老年急性大量硬膜下血肿钻孔引流术1例

正1病例资料84岁女性,因头部外伤后昏迷1 h入院。入院体格检查:神志浅昏迷,GCS评分7分,左枕部头皮肿胀,双侧瞳孔等大、等圆,直径3.0 mm,对光反射均灵敏,颈部略抵抗,双肺呼吸音粗,可闻及散在湿性啰音,左侧Babinski征阳性。既往有高血压病2级(高危)、冠心病、心力衰竭、心功能Ⅲ级5年,平时规律服用肠溶阿司匹林(0.1g,1次/d)、单硝酸异山梨酯片     

微创穿刺治疗慢性硬膜下血肿64例临床分析

我院自2002-04～2009-04应用北京万特福公司生产的YL-1型颅内血肿粉碎穿刺针治疗慢性硬膜下血肿64例,创伤小,安全可靠,疗效非常满意,现报告如下.     

慢性硬膜下血肿骨化并发恶性脑膜瘤1例

脑膜瘤起源于蛛网膜颗粒上的内皮细胞和成纤维细胞,因此脑膜瘤的好发部位基本与蛛网膜颗粒在脑膜上的分布情况一致。脑膜瘤的成因尚不完全明确,但部分病例与颅脑外伤有一定关系。我们报道1例与创伤密切相关的脑膜瘤病例：     

慢性硬膜下血肿伴钙化1例报告

患者,男,49岁,因发作性意识丧失2d伴四肢抽搐1次入院。3年前曾有头部外伤史,未予诊治。查体：体温：36．5℃、脉搏：65次／min、呼吸：18次／min、血压：120／75mmHg,GCS评分：15分,神清,言语清晰,双侧瞳孔等大等圆,直径3mm,光反射敏感,四肢肌力均为Ⅴ级,肌张力正常,双侧巴宾斯基征（-）。头颅CT示：左侧额颞顶部囊性占位,周围伴蛋壳样钙化,中线明显右移,     

Idiopathic chronic subdural hematoma in the newborn: case report

Background Evidence of intrauterine development of chronic subdural hematoma (CSH) is extremely rare. A very limited number of cases with known and acceptable etiological explanation are reported. We were able to find out only four presented cases of intrauterine development of CSH without known etiology.Case report We present our case with a newborn baby with CSH without known etiology. Short literature review and possible etiological factors are observed.     

A case of divergence palsy associated with bilateral chronic subdural hematoma

A surgical case of bilateral chronic subdural hematoma presenting with divergence palsy is reported. A 75-year-old female was admitted to hospital for investigation of 2-week history of diplopia. The initial neurological examination demonstrated no abnormal findings except Hess chart suggesting bilateral concomitant strabismus or paresis of lateral rectus on both eyes. Because of the homonymous diplopia disappeared when the object of fixation in brought closer and the patient showed no impairment of gaze movements and following movements, the patient was diagnosed as divergence palsy and referred to the Neurosurgical Service. The physical examination revealed a 75-year-old, alert, fully oriented with diplopia at distant gaze. A computerized tomographic (CT) scan showed bilateral subdural hematoma. Emergency burrhole evacuation was performed and total amount bilateral hematoma was 220ml. Diplopia disappeared postoperatively. Hess chart examined two months after the operation shows no impairment of divergence function. This is the first reported case of divergence palsy associated with bilateral chronic subdural hematoma in CT era.     

Calcified chronic subdural hematoma mimicking calvarial mass: a case report

Head trauma is a frequent cause of mortality and morbidity in the pediatric population. Chronic subdural hematoma is the most common traumatic lesion in head injury. Chronic subdural hematomas are rare in children older than 2 years old; they are more frequent during adolescence. Calcified or ossified chronic subdural hematoma is a rare entity that usually presents as a space-occupying lesion over the cerebral convexity. Chronic calcified subdural hematomas are reported less than chronic subdural hematomas. In this article, we report a successfully treated patient with surgical removal case of calcified chronic subdural hematoma mimicking calvarial mass.     

Parkinsonism caused by chronic subdural hematoma. A case report

A 48 year-old man with chronic subdural hematoma presented with a parkinsonian syndrome. Removal of the hematoma was followed by gradual disappearance of the extrapyramidal symptoms, as in other, rare, published cases.

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Sommario Gli Autori riportano il caso di un uomo di 48 anni affetto da ematoma cronico subdurale e che presentava una sindrome parkinsoniana. Come per le altre rare osservazioni presenti nella letteratura, alla rimozione dell'ematoma era seguita la graduale risoluzione dei sintomi extrapiramidali.

     

Essential thrombocythemia associated with subdural hematoma and postoperative intracerebral hemorrhage--a case report

A case of essential thrombocythemia (ET) associated with subdural hematoma and postoperative intracerebral hemorrhage was reported. A 57-year-old man had complained headache in the morning. Six hours later he was found unconsciousness and soon he was brought to our hospital. On admission he was comatose. There was no evidences of head injury and the X-rays were normal. A computed tomography (CT) scan revealed an acute subdural hematoma over the left temporoparietal region. Laboratory data revealed thrombocytosis of 85.7 x 10(4)/mm3 with increased red and white blood cell counts. Emergent right craniotomy was performed and a subdural clot was evacuated. Neither cortical damage nor vascular malformations were seen on the cortical surface. But a spurting cortical artery with a pin-hole could be seen. A postoperative CT scan revealed an intracerebral hemorrhage deep in the right hemisphere. Cerebral angiograms revealed no vascular anomalies. Postoperatively, the platelet count remained high and laboratory data including bone marrow finding, neutrophil alkali-phosphatase score and chromosome analysis were consistent with the diagnosis of essential thrombocythemia. The mechanisms of subdural hematoma formation and postoperative intracerebral hemorrhage associated with essential thrombocythemia were discussed.     

Generalized chorea associated with bilateral chronic subdural hematoma

A variety of acquired disorders cause chorea, such as hypoxia-ischemia injury, drugs, toxins, infections, autoimmune disorders, and endocrine and electrolyte abnormalities, but rarely subdural hematoma. We report the case of a 76-year-old woman who presented with sudden onset of generalized choreiform movements 1 day prior to admission. Computed tomography revealed a bilateral lateral convexity subdural effusion, which was later confirmed by magnetic resonance imaging as a chronic subdural hematoma. After surgical evacuation of the subdural hematoma, the choreoathetoid movements ceased completely. We propose that transient ischemia in the bilateral subcortical regions was from compression and distortion of the brain by the bilateral hematomas and resulted in generalized chorea.     

A case of infected subdural hematoma following chronic subdural hematoma irrigation]

A 71-year-old female complicating Parkinson disease and diabetes mellitus was admitted to our medical center for urinary tract infection one month after burr hole irrigation and drainage of left chronic subdural hematoma. Klebsiella pneumonia was detected in the bacterial culture of her urine. As antibiotic therapy started, her condition and peripheral white blood cell counts were improved. But consciousness level got worsened and right hemiparesis appeared. A CT scan showed re-accumulation of left subdural fluid so an emergent irrigation was performed. The old hematoma with slightly yellowish, bloody purulent fluid was found and an intracapsular drain was inserted. Klebsiella pneumonia was detected from the bacterial culture of the hematoma. After 10 days, a CT scan showed subdural fluid collection again, so, total hematoma capsule removal was performed. After the operation, her neurological and serological condition improved and she was discharged without any neurological deficits. Klebsiella pneumonia existing urinary tracts rarely infected subdural hematoma cavity via hematogenous dissemination. We must keep in mind this complication may occur when we see compromised host like this case. Total removal of infected hematoma capsule is considered as radical treatment.