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电影《猩球崛起》从人类在丛林中的猎猿行动开始,以扣人心弦的故事探讨科技进步与人类文明及生物进化之间的冲突。基因科学家威尔致力于研究出治疗老年痴呆症的药物,将新药在黑猩猩“明眸”身上做实验,发现能够刺激其大脑细胞进行自我修复,大幅度提高其智力。明眸产子后母性爆发,袭击了工作人员而被射杀。威尔将新生的小猩猩带回家,并给他起名凯撒。 相似文献
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日本科学家发现,有规律地饮用绿茶,也许能在人变老时延缓大脑老化,降低人患老年痴呆症的风险。据最新一期《美国临床营养学杂志》报道,日本东北大学研究人员对1 003位70 岁以上的日本老人进行了问卷调查,问卷问题涉及这些老人前 1个月的所有饮食、健康情况及生活习惯。老人们还接受了有 相似文献
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2006年11月19日,国际著名学术期刊《自然-医学》网络版在线发表了中科院上海生命科学研究院生物化学与细胞生物学研究所裴钢院士研究组关于β淀粉样蛋白产生过程新机制的最新研究成果。他们发现,β2-肾上腺素受体被激活后,会增强γ-分泌酶的活性,进而能够增加导致老年痴呆症即阿尔采末病(Alzheimer’s disease,AD)的β淀粉样蛋白的产生。 相似文献
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在科学技术飞速发展的今天,许多近乎于“幻想”的研究都变成了现实。目前,医学领域的科学家正在致力于研究一种可以再造“思想”的大脑植入芯片。这种芯片能够治愈从老年痴呆症到心不在焉等各种脑部异常,并且减轻大脑随着年龄而老化和失去记忆的严重程度。在将来,就像计算机出现的小故障一样,失忆问题也能轻松解决。 相似文献
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It has been estimated that Alzheimer's disease (AD), the most common form of dementia, will affect approximately 81 million individuals by 2040. To date, the actual cause and cascade of events in the progression of this disease have not been fully determined. Furthermore, there is currently no definitive blood test or simple diagnostic method for AD. Considerable efforts have been put into proteomic approaches to develop a diagnostic blood test, but to date these efforts have not been successful. More recently, there has been a stronger focus on lipidomic studies in the hope of increasing our understanding of the underlying mechanisms leading to AD and developing an AD blood test. It is well known that the strongest genetic risk factor for AD is the e4 variant of apolipoprotein E (APOE). Evidence suggests that the ApoE protein, a major lipid transporter, plays a key role in the pathogenesis of AD, and its role in both normal and aberrant lipid metabolism warrants further extensive investigation. Here, we review ApoE-lipid interactions, as well as the roles that lipids may play in the pathogenesis of AD. 相似文献
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Guangchun Han Jiya Sun Jiajia Wang Zhouxian Bai Fuhai Song Hongxing Lei 《基因组蛋白质组与生物信息学报(英文版)》2014,(4):156-164
Neurological disorders comprise a variety of complex diseases in the central nervous system, which can be roughly classified as neurodegenerative diseases and psychiatric disorders. The basic and translational research of neurological disorders has been hindered by the difficulty in accessing the pathological center (i.e., the brain) in live patients. The rapid advancement of sequencing and array technologies has made it possible to investigate the disease mechanism and biomarkers from a systems perspective. In this review, recent progresses in the discovery of novel risk genes, treatment targets and peripheral biomarkers employing genomic technologies will be dis- cussed. Our major focus will be on two of the most heavily investigated neurological disorders, namely Alzheimer's disease and autism spectrum disorder. 相似文献
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《现代生物医学进展》2015,(1):202
<正>增加脑部活动可以帮助具有老年痴呆症症状的老年人抵消与该病相关的负面变化,发表在《自然-神经科学》上的一项研究得出上述结论。老年痴呆症与大脑内的β-淀粉样蛋白(Aa)的累积有关,该累积会导致大脑细胞的损伤。William Jagust等人利用功能性核磁共振成像技术追踪健康年轻人、健康老年人以及大脑内已确定含有Aa沉淀的老年人这三组人在尝试记忆场景图片时,其各自大脑的活动情况。两组老年人完成试验的情况相当,但是在年轻人和脑内Aa累积的老年人之间的比较显示,大脑内视觉和 相似文献