试论安定性不合格消石灰对柔性路面质量的影响

由于过烧石灰的存在和消解不当等原因，造成消石灰安定性不合格，当石灰稳定类路面结构成型或投入使用后，消石灰中的生石灰颗粒开始吸收周围水分，慢慢消解，消解过程中发生体积膨胀，对路面产生破坏。解决石灰安定性问题对防止公路前修后坏，延长路面使用寿命具有积极的现实意义。     

电弧炉采用石灰石单渣冶炼工艺的研究

采用优质石灰石取代质量差的白灰 ,进行了单渣电弧炉熔炼。结果表明 ,钢水成本降低 ,钢水质量提高 ,取得了良好的效果。     

一种基于TDA16846的新型有源功率校正电路设计

介绍一种由集成电路芯片TDA16846完成的利用电荷泵(Charge Pump)实现的有源功率因数校正(APFC)技术。这种APFC电路的突出优点就是结构简单,通过对常规缓冲电路进行合理调整,可以很容易变成具有PFC功能的高性能开关电源。分析了APFC的工作原理,应用专用芯片TDA16846设计了电路图,并阐述了主要元件的功能。经实验证明,推荐电路设计合理,应用前景广阔。     

国际检验方法对促进产品质量提高的启示

对氨基二苯胺是性能优异的对苯二胺类防老剂的中间体。其质量好坏直接关系到防老剂4020、4010NA、688等品种的质量。对氨基二苯胺质量检验方法的提出则有助于我们开拓思路，更新质量观念，地症下药，分析原因，采取措施，全面提高其内在质量。     

主动约束层阻尼板结构动力学建模研究

将有限元法和GHM法相结合，给出了建立主动约束层阻尼（ACLD）板结构动力学模型的新方法。建模时，考虑到粘弹材料（VEM）的纵向位移影响，采用GHM方法描述VEM的本构关系，解决了VEM的力学特性随温度和频率变化的难点，避免因VEM的复杂本构关系而产生的微分积分方程。算例表明本文给出的建模方法是准确的，ACLD结构能够有效控制结构振动。     

纤维素酶分子的计算机辅助序列分析

利用生物大分子数据库（EMBL、SWALL和PDB）及软件技术（DNASIS和AntheProt）对嗜热子囊菌（Thermoascus aurantiacus)的产生的一种纤维素酶分子进行了序列分析及结构预测；通过多种来源的纤维素酶的比较研究，分析了其催化区、活性位点及局部保守序列。     

挡土墙填土曲线破裂面的地震主动土压力分析

在地震作用影响下，墙后填土处于极限平衡状态时，假定破裂面为一条通过墙趾的旋轮线。从墙后土楔中取出一微薄水平层，研究其在水平惯性力下的平衡，得到的地震土压力分布和土压力合力作用点位置均与实验结果相吻合。     

InGaAsP/InP有源双稳放大器的实验研究

本文报导了室温下F—P型InGaAsP/InP双异质结半导体激光放大器的双稳态效应,放大器的增益为20dB。     

CAD OF MILLIMETER WAVE OSCILLATORS WITH THE FDHB TECHNIQUE

In this paper the modelling, analysis and optimization of millimeter wave oscillatorsare investigated by using the a frequency-domain harmonic balance technique (FDHB), where theexternal-circuit impedances looking outside from the active device are calculated with a combinedtechnique of modes expansion, Galerkin, and collocation methods. The optimization results arein agreement with the experimental ones, which show the reliability of the presented model andoptimization.     

Structure-activity studies of human tumour necrosis factors

The mechanism by which tumour necrosis factors (TNF and lymphotoxin,also called TNFß and TNFß respectively) exerttheir cytotoxic activity on many malignant cells, remains largelyunknown. Furthermore, the broad array of differentiation (geneinduction) and mitogenic activities towards many primary cellsis still a subject of intensive investigation. TNF is an importantmediator in inflammation, immune responses and infection-relatedphenomena and these activities contribute to the severe toxidtyseen when TNF is used as an anticancer agent. The first stepin the mechanism of action is the specific binding of the ligandto its receptors and dissection of the molecular mechanism involvedin this interaction is the subject of this review. The reasonsfor the interest in this aspect are obvious: first, the developmentof strong antagonistic TNF analogues can be useful in dampeningthe potentially lethal or debilitating effects of an overproductionof the cytokine (as in septic shock or rheumatoid arthritis).Secondly, since two distinct TNF receptors exist, constructionof TNF muteins that distinguish between both types may leadto derivatives of this plekrtropic agent with a more restrictedbiological activity pattern. Ideally, one would like to developa TNF mutant that has retained its cytotoxic action on tumourcells without inducing the deleterious systemic toxteity. Suchan optimized TNF molecule could become a potent anticancer agent