Reliability-based hybrid ARQ as an adaptive response to jamming

We consider the use of selective-retransmission automatic repeat request (ARQ) protocols to improve communication performance on partial-time jamming channels. The schemes that we propose are based on reliability-based hybrid ARQ (RB-HARQ), in which the set of bits to be retransmitted is adaptively selected based on the estimated a posteriori probabilities at the output of a soft-input, soft-output decoder. For channels with bursty partial-time jamming, the RB-HARQ techniques are particularly appropriate because these techniques can identify which symbols need to be retransmitted, and the bursty nature of the jamming reduces the overhead required to relay this information to the source. We extend our reliability measure to incorporate not only a posteriori probability information but also estimates of the probability that a bit was jammed. We compare the performance of the proposed scheme with that of conventional approaches, in which a predetermined set of bits is retransmitted in response to a packet failure. The results show that RB-HARQ schemes can achieve higher throughput than these conventional approaches in several scenarios.     

Group 1 metabotropic glutamate receptors contribute to slow-onset potentiation in the rat CA1 region in vivo

It has been demonstrated in the CA1 region of the hippocampus in vitro, and in the dentate gyrus and CA1 region in vivo, that application of the metabotropic glutamate receptor (mGluR) agonist, 1S, 3R-amino cyclopentane 2,3-dicarboxylic acid triggers a slow-onset potentiation of synaptic transmission in the hippocampus. This study examined the involvement of group 1 and 2 mGluRs in this phenomenon in the CA1 region of freely moving rats. Drugs were applied via the lateral cerebral ventricle, and measurements were obtained from the CA1 region via permanently implanted electrodes. The group 1 mGluR agonists, 3,5-dihydroxyphenylglycine (DHPG, 20-100 nmol/5 microl) and trans-azetidine-2,4-dicarboxylic acid (ADA, 100 nmol-1 micromol/5 microl) induced a dose-dependent potentiation of basal synaptic transmission. The mGluR antagonist R,S-alpha-methyl-carboxyphenylglycine (MCPG, 1 micromol), and the group 1 mGluR antagonist, S-4-carboxyphenylglycine (4CPG, 100 nmol) completely inhibited the effects of both DHPG and ADA. The group 2 mGluR agonist, (S)-4-carboxy-3-hydroxy phenylglycine (4C3H-PG, 50-200 nmol/5 microl) induced a dose-dependent decrease of basal synaptic transmission. These results suggest that in the CA1 region in vivo, slow-onset potentiation may be mediated by group 1 mGluRs.     

Smoking cessation among self-quitters.

Examined cessation among 630 smokers who quit abruptly on their own. Continuous, complete abstinence rates were 33% at 2 days, 24% at 7 days, 22% at 14 days, 19% at 1 mo, 11% at 3 mo, 8% at 6 mo postcessation, and 3% at 6 mo with biochemical verification. Slipping (smoking an average of less than 1 cigarette/day) was common (9% to 15% of Ss) and was a strong predictor of relapse; however, 23% of long-term abstainers slipped at some point. Results challenge beliefs that most smokers can initially stop smoking and that most relapse occurs later on postcessation. (PsycINFO Database Record (c) 2010 APA, all rights reserved)