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1.
获得性免疫缺陷综合征(AIDS) 血红蛋白(rib) 反转录聚合酶链反应(RT—PCR) 骨形成蛋白(BMP) 白细胞介素(IL) 扫描电镜(SEM) 计算机辅助制作(CAM) 磁共振成像(MRI) 颞下颌关节紊乱病(TMD)  相似文献   

2.
目的评估不同实验条件下树脂水门汀与氧化锆瓷之间的剪切粘接强度(SBS)。 方法使用计算机辅助设计与制作(CAD/CAM)设备切割并烧结制作6个边长为2 cm的正方体氧化锆试件。按不同实验条件将8种双固化树脂水门汀分为13组(n = 20):(1)Monobond N + Multilink N(MMN组);(2)Ivoclean + Monobond N + Multilink N(IMM组);(3)Ivoclean + Monobond N + Multilink N[无喷砂,IMM(ns)组];(4)Tetric N-Bond Universal + Multilink Speed(TUM组);(5)Multilink Speed(MLS组);(6)Z-Prime Plus + TheraCem(ZPT组);(7)Z-Prime Plus + Duo-Link(ZPD组);(8)Single Bond Universal + RelyX U200(SRU组);(9)RelyX U200(RXU组);(10)Single Bond Universal + RelyX Ultimate Clicker(SRU组);(11)OptiBond Versa + Kerr NX3(OVK组);(12)Clearfil Universal Bond + Clearfil SAC(CUS组);(13)Clearfil SAC(SAC组)。将每组树脂水门汀自混合注入透明模具并将其无压力置于氧化锆面后进行光照固化。所有试件在37 ℃水中存储24 h后进行SBS测试。采用SPSS 19.0软件One-Way ANOVA(P<0.05)对SBS进行统计学分析。利用电子体视显微镜观察氧化锆端断裂界面。 结果本实验中13组树脂水门汀的SBS(单位:MPa)降序排列依次为:OVK(27.51 ± 3.65)>IMM(27.28 ± 3.79)>SRC(26.77 ± 3.62)>CUS(25.36 ± 3.10)>TUM(25.22 ± 4.88)>ZPD(23.96 ± 6.25)>MLS(23.13 ± 2.74)>MMN(23.07 ± 3.71)>SAC(22.20 ± 3.59)>IMM(ns)(21.99 ± 3.50)>SRU(19.19 ± 2.27)>ZPT(18.62 ± 2.08)>RXU(15.04 ± 4.02)。 结论(1)不同树脂水门汀的SBS具有材料依赖性;(2)在氧化锆表面进行喷砂处理、使用专用清洗剂或将通用型粘接剂作为底漆使用有利于提高树脂水门汀的SBS。  相似文献   

3.
(说明:以下词汇在论文中首次出现时应注中文全称,括号内直接写缩略语)获得性免疫缺陷综合征(AIDS) 骨形态生成蛋白(BMP) 计算机辅助设计(CAD) 计算机辅助制作(CAM) 计算机体层摄影术(CT) 血红蛋门(Hb) 人类免疫缺陷病毒(HIV) 白细胞介素(IL)  相似文献   

4.
获得性免疫缺陷综合征(AII)S) 骨形态生成蛋白(BMP) 计算机辅助设计(CAD) 计算机辅助制作(CAM) 计算机体层摄影术(CT) 血红蛋白(Hb)获得性免疫缺陷综合征(AIDS)  相似文献   

5.
(说明:以下词汇在论文中首次出现时应注中文全称,括号内直接写缩略语) 获得性免疫缺陷综合征(AIDS)血红蛋白(Hb)反转录聚合酶链反应(RT-PCR)骨形成蛋白(BMP)白细胞介素(IL)扫描电镜(SEM)  相似文献   

6.
获得性免疫缺陷综合征(AIDS) 骨形成蛋白(BMP) 计算机辅助制作(CAM) 计算机辅助设计(CAD) 计算机体层摄影术(CT)  相似文献   

7.
获得性免疫缺陷综合征(AIDS) 骨形成蛋白(BMP) 计算机辅助制作(CAM) 计算机辅助设计(CAD)  相似文献   

8.
目的:探讨基牙颈缘线曲率对CAD/CAM全瓷冠及金属烤瓷冠边缘适合性的影响。方法与材料:在模型的上颌中切牙上预备3种类型的基牙(曲率1mm、3mm、5mm)。每种类型的基牙分别制作5个全瓷冠(Cercon system.Degudent)和5个金属烤瓷冠。采用Two—way ANOVA和Tukey—HSD检验(α=O.05)计算和分析冠边缘的适应性。结果:全瓷冠唇侧、舌侧、近中和远中的平均边缘缝隙(SD)无统计学差异.分别是:曲率为1mm的全瓷冠边缘缝隙为54(10)、51(11)、47(13)、49(9)岬;曲率为3mm的全瓷冠边缘缝隙为49(12)、53(11)、54(10)、55(12)岬:曲率为5mm的全瓷冠边缘缝隙为57(12)、54(11)、53(10)、52(9)μm。曲率为1mm的烤瓷冠唇侧、舌侧、近中和远中的平均边缘缝隙(SD)值分别是36(7)、41(9)、26(8)、28(10)邮.曲率为3mm的烤瓷冠唇侧、舌侧的平均边缘缝隙(SD)值为45{8).48(9)μm.均显著大于近中(P=O.01和0007)和远中(P=0.03和0.02)的边缘缝隙。曲率为5mm的烤瓷冠唇侧、舌侧平均边缘缝隙(SD)值为76(10)、74(15)μm.均显著大于近中(P=O001和0.001)和远中(P=0.001和0001)的边缘缝隙。结论:基牙的颈缘线曲率对全瓷冠的边缘适合性无显著影响.但对烤瓷冠的边缘适合性有显著影响。  相似文献   

9.
2010年度共有74本口腔医学杂志被SCI收录,影响因子(IF)排在前6位的杂志为J CLIN PERIODONTOL(3.933)、J DENT RES(3.773)、J ENDODONT(3.291)、CARIES RES(2.926)、DENT MATER(2.920)和ORAL ONCOL(2.871)。  相似文献   

10.
本文报告1例咽旁滑膜肉瘤,患者男,15岁,以左咽旁无痛性逐渐增生包块1月就诊。专科检查发现左侧咽旁有一大小约8 cm×4 cm×3 cm肿物,蒂部位于左扁桃体上隐窝。影像学检查见左侧口咽不规则肿块,与左舌根后份分界不清,增强扫描明显不均匀强化。免疫组织化学检查:上皮细胞膜抗原(+),细胞角蛋白(CK)19(+),CD7(+),波形蛋白(+),CK10(-),钙黏蛋白(+),B淋巴细胞瘤-2(-),CD2(-),CD10(-),CD138(+),CD99(+),白细胞共同抗原(+),Ki-67(20%+)。病理诊断为咽旁滑膜肉瘤。  相似文献   

11.
妊娠期牙龈瘤是妊娠期女性由于性激素水平升高导致牙龈慢性炎症加重,从而在牙龈局部形成的瘤样病变,多见形成单个龈瘤,而多发性者少见。本文报道1例罕见的妊娠期多发性牙龈瘤,患者在行全口洁治后未行其他特殊治疗,病变大小未见明显改变,于分娩后5个月内龈瘤全部自行消退。  相似文献   

12.
目的:为评价瘤体结扎加口腔微电刀治疗孕妇妊娠性牙龈瘤临床效果。方法:对69名孕妇牙龈瘤患者,根据瘤体大小分为单发和多发型,采用简单,分段和悬吊结扎后加微电刀切除的微创疗法。结果:69例均取得不同程度治疗效果,其中单发型孕瘤治愈率,基本治愈率合计94.44%,好转率5.56%;多发型治愈率,基本治愈率合计72.22%,好转率26.67%;经卡方检验两型妊娠性牙龈瘤不同疗效之间有高度显著性差异(P=0.003),单发型治疗效果更优。结论:通过瘤体结扎阻断和减缓瘤体的血液供给,使瘤体缩小后再采用口腔微电刀切除,两术起到协同作用,是一种微创和安全有效的治疗方法,值得在孕妇口腔治疗中推广应用。  相似文献   

13.
Congenital epulis is a rare benign hamartoma of the alveolar ridge found in the newborn. The clinical characteristics of this lesion show some variability, and opinion of its pathogenesis lacks consensus. Congenital epulis can cause feeding or respiratory compromise. A case report and review of the literature is presented. The diagnosis, pathology, and treatment are reviewed.  相似文献   

14.
The purpose of this study was to describe an alternative surgical approach to removing epulis while preserving and improving the mucogingival complex. After scaling and root planing, six patients underwent surgical treatment to remove the growths. A subepithelial connective tissue graft was interposed between the bone and epulis using a pedicle flap. The lesion was always preserved. The epulis disappeared after several months, with minor variations in healing time among patients. At 60-month follow-up visits, all patients exhibited good gingival health, with no recurrence.  相似文献   

15.
Congenital epulis is not a common lesion. 2 rare neonatal multiple cases are reported. The treatment of choice of these lesions is radical excision during early infancy.  相似文献   

16.
Congenital granular cell lesion, or congenital epulis, is a rare lesion of the newborn. The sight of a newborn infant with congenital epulis can be startling for both parents and healthcare professionals. The lesion appears most commonly in the maxillary alveolar ridge, with predominance in females. The etiology and histogenesis of the lesion remain obscure. This lesion can pose a risk of airway obstruction or can interfere with feeding. The report presented here describes a case of congenital granular cell lesion in the left maxillary region. The lesion was causing a feeding problem and, hence, was excised under local anesthesia.  相似文献   

17.
An unusual case of a double congenital epulis involving the alveolar mucosa of both the maxilla and mandible is reported. The authors discuss the clinical picture, histological findings, etiology and treatment of this rare lesion.  相似文献   

18.
19.
The congenital granular-cell epulis is an uncommon pedunculated tumor of the alveolar mucosa found only in newborn infants. This lesion can present an alarming appearance and interfere with efforts at nursing. The generally accepted treatment is excision through the pedicle, and recurrence is not to be anticipated. An example of a tumor fulfilling the clinical and histopathologic criteria of this lesion is reported.  相似文献   

20.
An accumulation of granular cells associated with the enamel organ of a developing deciduous, incisor tooth was noted during light microscopy (LM) of serial sections from the right half of a mandible from a stillborn, female infant of 37 weeks gestations. There was a break in continuity of the enamel matrix associated with the focus of granular cells, which appeared to be continuous with, and arise directly from, the cells of the stratum intermedium.
The granular cells were very similar histologically to those of a congenital epulis but it is impossible to say whether the lesion would have developed sufficiently to present clinically as an epulis. Less probably, the granular cells might eventually have been associated with the subsequent development of a granular cell ameloblastoma or ameloblastic fibroma. If the lesion does represent an early stage in the formation of a congenital epulis, it would support the odontogenic theory of origin of the granular cells.  相似文献   

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